Fig. 12From: Heart-targeted amelioration of sepsis-induced myocardial dysfunction by microenvironment responsive nitric oxide nanogenerators in situMyocardium-targeted NO release effectively prevented mitochondrial dysfunction in the myocardium of the LPS-induced sepsis model. a Expression of mitochondrial mediator proteins Drp1 and Mfn 2. b, c Quantitative analysis of Drp1and Mfn 2 expression (n = 8). Data were presented as mean ± SD, *P < 0.05; **P < 0.01. d TEM analysis of myocardial sections (scale bar = 1 μm)Back to article page