Fig. 1From: Biomimetic doxorubicin/ginsenoside co-loading nanosystem for chemoimmunotherapy of acute myeloid leukemiaCharacteristic and anti-tumor mechanism of the biomimetic nanosystem (DR@PLip). A Preparation and anti-tumor mechanism of DR@PLip. Lipsomes co-loading DOX and Rg3 were co-extruded with PM to construct the DR@PLip. The biomimetic nanosystem in blood encountered the AML cells and induced powerful immunogenic cell death. Then the activated effector T cells penetrated deep in bone marrow to kill the AML cells, as well as eliminated the AML cells remained in blood. B Size distribution and zeta potential of the nanosystem. C TEM images of the nanosystem. Scale bar, 100Â nm. D The PLT relative protein detected in different samplesBack to article page