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Fig. 7 | Journal of Nanobiotechnology

Fig. 7

From: Neutrophil membrane engineered HucMSC sEVs alleviate cisplatin-induced AKI by enhancing cellular uptake and targeting

Fig. 7

NEX promotes NRK52E cellular uptake and their proliferation, and decreases cisplatin-induced levels of oxidative stress. a Laser scanning confocal microscopy was used to detect the internalization of DIO-labeled hucMSC-sEVs and NEX by NRK52E cells at different time points (×600). b An automatic microplate reader was applied to determine the level of oxidative stress in NRK52E cells treated with cisplatin and three kinds of vesicles (×40) and c to evaluate the cell proliferation activity of NRK52E cells after cisplatin and three kinds of vesicle administration (×40). d qRT-PCR was used to detect the expression levels of proinflammatory and anti-inflammatory factors in NRK52E cells treated with three kinds of vesicles and LPS

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