Fig. 4

The mRNA expression of metabolism-related genes of AML12 cells and Hepa1–6 cells (a); Western blot analysis of HK-II, c-Myc and GAPDH. β-actin was used as an internal reference (b); The activity of α-KGDH (c); The Western blot analysis of TBK1 and STING in AML12 cells and Hepa1–6 cells (d); Effects of BCP NPs, ABZI and BCP NPs + ABZI treated Hepa1–6 cells triggered dimerization of STING (e); Effects of BCP NPs, ABZI and BCP NPs + ABZI treated Hepa1–6 cells triggered phosphorylation of TBK1 in AML12 cells (f) and Hepa1–6 cells (g); Different concentration BCP NPs treated Hepa1–6 cells triggered phosphorylation of TBK1 in Hepa1-6 cells (h); The relatived production of IFN-β after the treatment of different concentration BCP NPs treated Hepa1–6 cells (i)